Hasil untuk "hep-lat"

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arXiv Open Access 2024
Theory on CKM and heavy quark decay

Oliver Witzel

The combination of precise experimental measurements and theoretical predictions allows to extract Cabibbo-Kobayashi-Maskawa (CKM) matrix elements or constrain flavor changing processes in the standard model. Focusing at theoretical predictions, we review recent highlights from the sector of heavy charm and bottom quark decays. Special emphasis is given to nonperturbative contributions due to the strong force calculated using lattice QCD.

en hep-ph, hep-lat
arXiv Open Access 2013
Topology across the finite temperature transition studied by overimproved cooling in gluodynamics and QCD

V. G. Bornyakov, E. -M. Ilgenfritz, B. V. Martemyanov et al.

Gluodynamics and two-flavor QCD at non-zero temperature are studied with the so-called overimproved cooling technique under which caloron solutions may remain stable. We consider topological configurations either at the first occuring stable plateau of topological charge or at the first (anti)selfdual plateau and find the corresponding topological susceptibility at various temperatures on both sides of the thermal transition or crossover. In pure gluodynamics the topological susceptibility drops sharply at the deconfinement temperature while in full QCD it decreases smoothly at temperatures above the pseudocritical one. The results are close to those calculated by other methods. We interpret our findings in terms of the (in)stability of calorons with non-trivial holonomy and their dyon constituents against overimproved cooling.

en hep-lat, hep-th
arXiv Open Access 2012
Cooling study of Dirac sheets in SU(3) lattice gauge theory below T_c

E. -M. Ilgenfritz, B. V. Martemyanov, M. Müller-Preussker

Using a standard cooling method for SU(3) lattice gauge fields constant Abelian magnetic field configurations are extracted after dyon-antidyon constituents forming metastable Q=0 configurations have annihilated. These so-called Dirac sheets, standard and non-standard ones, corresponding to the two U(1) subgroups of the SU(3) group, have been found to be stable if emerging from the confined phase, close to the deconfinement phase transition, with sufficiently nontrivial Polyakov loop values. On a finite lattice we find a nice agreement of the numerical observations with the analytic predictions concerning the stability of Dirac sheets depending on the value of the Polyakov loop.

en hep-lat, hep-ph
arXiv Open Access 2009
Quark electric dipole moment induced by magnetic field

P. V. Buividovich, M. N. Chernodub, E. V. Luschevskaya et al.

We show numerically that quarks develop an electric dipole moment in the direction of a sufficiently intense magnetic field due to local fluctuations of topological charge. This anomalous CP-odd effect is a spin analogue of the Chiral Magnetic Effect in QCD.

en hep-ph, hep-lat
S2 Open Access 1988
Regulation of the expression of type 1 plasminogen activator inhibitor in Hep G2 cells by epidermal growth factor.

C. Lucore, S. Fujii, T. Wun et al.

To identify factors potentially influencing expression of type 1 plasminogen activator inhibitor (PAI-1), we characterized the human tissue-specific distribution of PAI-1 mRNA and the influence of epidermal growth factor (EGF) on expression of steady state levels of PAI-1 mRNA and secretion of PAI-1 by Hep G2 cells. Two species of PAI-1 mRNA (3.2 and 2.2 kilobases) were detected, and the ratio of the two varied among tissues (3 to 5:1) in contrast to the 1:1 ratio detected in Hep G2 cells. Expression of PAI-1 mRNA was inversely related to the distribution of tissue-type plasminogen activator mRNA (2.3 kilobases). Nu-Serum, a growth media supplement, increased steady state levels of PAI-1 mRNA 5-fold within 3 h. Factors responsible were found to be trypsin-sensitive and dialysis-resistant. Antisera to EGF attenuated Nu-Serum-induced increases of PAI-1 mRNA by 57%, suggesting that EGF or EGF homologous peptides contributed to the response. EGF elicited increases of PAI-1 mRNA levels in a dose-dependent manner. Induction was rapid (7-fold at 3 h with 5 ng/ml) and complete within 10 h. The response was not attenuated by cycloheximide (25 micrograms/ml). Factor X and glyceraldehyde-3-phosphate dehydrogenase mRNA did not increase. Increased levels of PAI-1 antigen were detected in conditioned media of Hep G2 cells by 4 h and were maximal at 8 h (6-fold). We conclude that the expression of PAI-1 mRNA is tissue-specific and regulated by epidermal growth factor in Hep G2 cells.

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